Harmful substances in cigarette smoke

SubstanceSpecific harmful effect
TarCarcinogen that triggers mutations in lung cells, potentially causing lung cancer. Also damages and paralyses cilia lining the airways.
NicotineAddictive stimulant that increases heart rate and blood pressure. Makes it difficult to stop smoking. Narrows blood vessels.
Carbon monoxideBinds irreversibly to haemoglobin, forming carboxyhaemoglobin. This reduces the oxygen-carrying capacity of the blood.
Irritant particlesStimulate excess mucus production in the airways and contribute to chronic bronchitis.

Emphysema and COPD

When tar damages and paralyses cilia, mucus and trapped pathogens are no longer swept away from the lungs. Infections become more frequent. The repeated inflammation and infection damage the delicate alveolar walls. In emphysema, alveolar walls break down, creating fewer, larger air spaces. This reduces the total surface area for gas exchange and makes breathing less efficient.

Patients with emphysema struggle to get enough oxygen into their blood and find it hard to exhale fully. Chronic bronchitis (persistent inflammation and mucus production in the bronchi) combined with emphysema is called chronic obstructive pulmonary disease (COPD).

Lung cancer

Tar is a carcinogen: it causes mutations in the DNA of lung cells. If mutations occur in genes controlling cell division, cells may divide uncontrollably, forming a tumour. If the tumour is malignant, it invades surrounding tissue and may spread to other parts of the body (metastasis).

Lung cancer is strongly linked to smoking: the longer and more heavily a person smokes, the greater their risk. Stopping smoking at any stage reduces the risk compared to continuing, though it remains higher than for someone who never smoked. Passive smoking (inhaling other people's smoke) also increases lung cancer risk.